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[h] General Pathology Flashcards

[i] Master this session in just 5 minutes.

[q] ……….. phase of wound healing is characterized by Clot formation, increase of vessel permeability and neutrophil migration into tissue; macrophages clear debris 2 days later.

[c]IFNob3cgbWUgdG hlIGFuc3dlcg==[Qq]

[f]IEluZmxhbW1hdG9yeS4=[Qq]

[q] ………. phase of wound healing is characterized by deposition of granulation tissue and type III collagen, angiogenesis, epithelial cell proliferation, dissolution of clot, and wound contraction (mediated by myofibroblasts).

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[f]IFByb2xpZmVyYXRpdmUu[Qq]

[q] ………. phase of wound healing is characterized by Type III collagen replaced by type I collagen, increase of tensile strength of tissue.

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[f]IFJlbW9kZWxpbmcu[Qq]

[q] Zinc deficiency cause delayed wound healing because ………… require zinc to function.

[c]IFNob3cgbWUgdG hlIGFuc3dlcg==[Qq]

[f]IENvbGxhZ2VuYXNlcy4=[Qq]

[q] Vitamin c deficiency causes delayed wound healing because ………… requires vitamin c for during collagen synthesis.

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[f]IHByb2xpbmUgYW5kIGx5c2luZSBoeWRyb3h5bGFzZS4=[Qq]

[q] ………. is a type of abnormal scar formation due to deposition of excess type III collagen parallel to the wound, Confined to borders of original wound, with low rate of recurrence.

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[q]………. is a type of abnormal scar formation due to deposition of excess type I and III collagen with disorganized organization, Extends beyond borders of original wound, with high rate of recurrence.

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[q] ………, ………, and ……… are growth factors that mediate Angiogenesis.

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[q] ……….. is an intracellular yellow-brown pigment associated with normal aging. Composed of polymers of lipids and phospholipids complexed with protein.

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[f]IExpcG9mdXNjaW4u

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[q] In primary amyloidosis, there is abnormal aggregation of ………. in multiple tissues and commonly seen in ………..?

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[f]IEFMIChmcm9tIElnIExpZ2h0IGNoYWlucyksIFNlZW4gaW4gcGxhc21hIGNlbGwgZGlzb3JkZXJz
CihtdWx0aXBsZSBteWVsb21hKS4=[Qq]

[q] In secondry amyloidosis, there is abnormal aggregation of ………. in multiple tissues and commonly seen in ………..?

[c]IFNob3cgbWUgdG hlIGFuc3dlcg==[Qq]

[f]IFNlcnVtIEFteWxvaWQgQSAoQUEpLCBTZWVuIGluIGNocm9uaWMgaW5mbGFtbWF0b3J5
CmNvbmRpdGlvbnMgKHJoZXVtYXRvaWQgYXJ0aHJpdGlzLCBJQkQsIGZhbWlsaWFsIE1lZGl0ZXJyYW5lYW4gZmV2ZXIsIHByb3RyYWN0ZWQgaW5mZWN0aW9uKS4=[Qq]

[q] In Dialysis-related amyloidosis, there is abnormal aggregation of ………. in multiple tissues and commonly seen in ………..?

[c]IFNob3cgbWUgdG hlIGFuc3dlcg==[Qq]

[f]IM6yMi1taWNyb2dsb2J1bGluLCBTZWVuIGluIHBhdGllbnRzIHdpdGggRVNSRCBhbmQvb3Igb24gbG9uZy10ZXJtIGRpYWx5c2lzLg==[Qq]

[q] In Alzheimer disease, there is abnormal aggregation of ………. in the brain?

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[f]IM6yLWFteWxvaWQgcHJvdGVpbi4=[Qq]

[q] In Isolated atrial amyloidosis, there is abnormal aggregation of ………. in the atria?

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[f]IEFOUC4=[Qq]

[q] In Systemic senile (agerelated) amyloidosis, there is abnormal aggregation of ………. in the ventricles?

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[f]IE5vcm1hbCAod2lsZC10eXBlKSB0cmFuc3RoeXJldGluIChUVFIpLg==[Qq]

[q] In Familial amyloid cardiomyopathy, there is abnormal aggregation of ………. in Ventricular endomyocardium?

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[f]IE11dGF0ZWQgdHJhbnN0aHlyZXRpbiAoQVRUUiku[Qq]

[q] In Familial amyloid polyneuropathies, there is abnormal aggregation of ………. in neurons?

[c]IFNob3cgbWUgdG hlIGFuc3dlcg==[Qq]

[f]IE11dGF0ZWQgdHJhbnN0aHlyZXRpbiAoQVRUUiku[Qq]

[q] Amyloid deposits is visualized by Congo stain and appear ………. on nonpolarized light,  and ………… on polarized light.

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[f]IHJlZC9vcmFuZ2UsIGFwcGxlLWdyZWVuIGJpcmVmcmluZ2VuY2Uu

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